It certainly debunks your claim that this is a new technology which hasn't been previously scrutinised for adverse health effects.
As does the long list of research papers
here.
Some examples:
Alfheim I, Ramdahl T,
Contribution of wood combustion to indoor air pollution as measured by mutagenicity in Salmonella and polycyclic aromatic hydrocarbon concentration,
Environ. Mutagen 1984;6(2):121-130
Abstract: Samples of airborne particles have been collected in the same room when the room was heated by electricity and when heating was done by woodburning. These samples were compared with respect to mutagenic activity and concentration of polycyclic aromatic hydrocarbons (PAH). The effects of the various heating conditions were examined in the presence and absence of tobacco smoking. Whereas wood heating in an "airtight" stove was found to cause only minor changes in the concentration of PAH and no measurable increase of mutagenic activity of the indoor air, both these parameters increased considerably when wood was burned in an open fireplace, yielding PAH concentrations comparable to those of ambient urban air. Relatively high concentrations of moderately polar polycyclic aromatic hydrocarbon derivatives were also found in the indoor air when wood was burned in an open fireplace. Woodburning in the closed stove did, however, result in increased concentrations of mutagenic compounds and PAH on particles sampled in the vicinity of the house. The effects of wood burning in an open fireplace on the mutagenic activity of indoor air could still be considered moderate when compared to those resulting from tobacco smoking in the room. The extracts of particles collected when moderate smoking occurred were several times more mutagenic than samples from urban air collected close to streets with heavy traffic when measured in the Salmonella assay with strain TA98 with metabolic activation. >$/p>
Butterfield, P, LaCava, G. Edumunston E, Penner, J. 1989.
Woodstoves and indoor air: the effects on preschooler’s upper respiratory symptoms. J. Environ. Health 52:172-73. (L&K, Ref 14)
Boone PM, Rossman TG, Daisey JM.
The genotoxic contribution of wood smoke to indoor respirable suspended particles.
Environment International 1989 15:361-368.
Browning KG, Koenig JQ, Checkoway H, Larson, TV, Peirson WE.,
A questionnaire study of respiratory health in areas of high and low ambient wood smoke pollution,
Pediatr. Asthma All. Immunol. 4:183-91, 1990
Collings DA, Sithole SD, Martin KS,
Indoor woodsmoke pollution causing lower respiratory disease in children,
Trop Doct, 1990 Oct;20(4):151-155
Abstract: Suggested aetiological factors were evaluated in 244 consecutive children presenting with lower respiratory disease at Marondera Hospital, Zimbabwe. Data obtained from these children were compared with information obtained from 500 children seen at the local well baby clinic. There were no differences in the prevalence of malnutrition, breast feeding, overcrowding, poor housing conditions and poverty in these two groups of children. A significant association was identified between lower respiratory disease and exposure to atmospheric woodsmoke pollution in young children. Air sampling within the kitchens of 40 children revealed levels of atmospheric pollution far in excess of the WHO recommended exposure limit. Elevated carboxyhaemoglobin concentrations confirmed childhood smoke inhalation. We suggest that in many Third World communities a chemical pneumonitis resulting from the inhalation of noxious constituents of woodsmoke predisposes to lower respiratory disease in children.